Alzheimer’s Disease, Prion Disease And Transmissible Spongiform Encephalopathy
Excluding the recent coronavirus pandemic, neurodegenerative disease is the fastest-growing cause of death on the planet. Alzheimer’s disease alone is taking the lives of 50-100 million people around the world now. Millions will die of the disease this year, while millions more will be diagnosed and misdiagnosed. Millions of additional people will go undiagnosed. The prion pandemic has been accelerating for years. It will never go away.
A variety of factors can trigger neurodegenerative disease, including genetics, head trauma and prions. Prions are an infectious, deadly protein that consumes the brain. Prion disease is clinically known as transmissible spongiform encephalopathy (TSE). As the name suggests, TSEs are transmissible via bodily fluids (milk, blood, saliva, mucus, urine, feces, tissue and skin).
Prions shed from infected humans via mucus and saliva, for example, are highly transmissible. Prion disease is always fatal. Thanks to misinformation and mismanagement, this aggressive and progressive brain disease has killed millions of people around the world over the past 40 years. It’s also killing livestock, wildlife and sea mammals. This pandemic has been gaining momentum for years.
Most people don’t believe that Alzheimer’s disease, Parkinson’s disease, Creutzfeldt-Jakob disease (CJD) and chronic traumatic encephalopathy (CTE) are infectious. When we press most neurologists for evidence, there is none. It’s all based on wishful thinking. In fact, all evidence confirms the contrary. Research can confirm the issue, but it has been blocked for years. Therefore, we are connecting the dots based on public information and expert research.
Supposedly, no one knows the scope of the prion problem. The evidence swept under the rug and the global misinformation campaign suggest otherwise. The pandemic is more severe in some countries than others. Finland and Iceland were at the top of the list just a few years ago. Now, countries in the Middle East and Persian Gulf states have soared to the top of the list.
The ongoing mismanagement of highly infectious forms of brain disease made it easy to predict the global failure with the coronavirus. Public health and science have taken a back seat on prion disease for decades. As such, mismanagement of the coronavirus will likely continue.
The Alzheimer’s Pandemic
Neurodegenerative disease has been surging around the world for the past 40 years. It parallels the global surge in autism. The reasons are insane.
Despite millions of deaths every year, experts suggest that the prevalence of Alzheimer’s disease will quadruple by 2050, if not sooner. Unfortunately, there is a growing stack of evidence that Alzheimer’s disease, Parkinson’s disease and other brain diseases are transmissible. Deadly, self-replicating proteins appear to be one of the common threads. Childhood cases of Creutzfeldt-Jakob disease are increasing and they used to be nonexistent.
In the United States, for example, deaths from Alzheimer’s disease increased 71 percent from 2000 to 2013. During that time, deaths from heart disease decreased 14 percent.
At $236 billion a year, Alzheimer’s disease is already the most expensive disease in the United States. Most countries are feeling the pinch on public budgets.
Alzheimer’s disease is surging at a rate of at least 15 percent per year in many countries–the largest increase of all major causes of death. Given the vast numbers of people who go undiagnosed, the real number is probably double the official statistics.
It accounted for 108,227 documented deaths (and thousands more of undiagnosed and undocumented ones) in the U.S. alone in 2015. A similar pattern is emerging around the globe–some regions much more than others. In the U.S., nearly one in every five Medicare dollars is spent on people with Alzheimer’s or another dementia. These costs will continue to increase sharply as baby boomers age and the prion contagion spreads, soaring to more than $1 trillion in 2050.
The Alzheimer’s/Parkinson’s epidemic is more widespread than anyone knows. Physicians have withheld millions of diagnoses from patients and their families. According to an investigative report by the Alzheimer’s Association, physicians in the U.S. only inform 45 percent of patients about their Alzheimer’s disease diagnosis. In addition to ethical concerns, such suppression helps shade the pandemic. Meanwhile, millions more go undiagnosed and misdiagnosed every year.
A groundbreaking study suggested that Alzheimer’s disease actually causes six times more deaths than official statistics indicate. In reality, the study said that Alzheimer’s disease was the underlying cause in more than 500,000 deaths in 2010. Unfortunately, Alzheimer’s deaths are often attributed to conditions, such as pneumonia. It appears that more accurate numbers make Alzheimer’s disease the third-leading cause of death in the United States, behind heart disease and cancer. Researchers at the Rush University Medical Center in Chicago led the study and published their results in 2013 in the medical journal Neurology.
Governments and industry are working diligently to keep prion disease off the public radar. The epidemic will persist. A cure does not exist. It’s virtually impossible to sterilize anything exposed to a person with prion disease. Hospital systems are now on high alert after several cases of prion exposure. Ignoring the truth about prions on a broader scale is making the pandemic worse.
The Prion Connection
To advance the conversation rapidly, I hypothesize that Alzheimer’s disease is a transmissible disease. It’s infectious. It’s contagious. It’s fatal. Pick the word that works for you.
- As part of the hypothesis, I suggest that Alzheimer’s disease is a form of prion disease. Prion is a term based on the name proteinaceous, infectious particle.
- I suggest that prion disease is a spectrum disease, where Alzheimer’s disease and Creutzfeldt-Jakob disease (CJD) are on the same spectrum. The primary difference between these two forms of prion disease is that they attack the same region of the brain at different speeds. CJD is highly infectious and clearly mismanaged. The same can be said of Alzheimer’s disease since no one knows where along the spectrum the disease becomes infectious. I suggest that they are infectious at all phases of the spectrum. Hospitals will dispose of equipment contaminated by CJD patients, while sending patients home to die, where they proceed to expose family and friends, while contaminating entire communities. CJD patients should be quarantined, but CJD isn’t even a reportable disease across the U.S. and most countries. Neurologists are just guessing when distinguishing between CJD and Alzheimer’s disease. Mismanagement of this entire spectrum is fueling a public health disaster.
- I suggest that Parkinson’s disease also is a prion disease. The difference between Alzheimer’s disease and Parkinson’s disease is that the prions are destroying different parts of the brain. In Alzheimer’s, the region of the brain responsible for memory is under attack. With Parkinson’s disease, the region of the brain responsible for movement is under attack. Otherwise, the prion dynamics are the same as with Alzheimer’s and CJD.
- As the prion disease pandemic explodes, municipal wastewater treatment systems are becoming weapons of mass destruction. They are collection points for prions in blood, saliva, mucus, urine, feces, tissue and skin. Since we now dump most of our sewage on crops, ranches, parks, playgrounds, golf courses and gardens, prions are threatening our food and water supplies.
- Prion disease is a threat to families, caregivers, health care systems and entire communities.
- As the prion disease epidemic explodes among humans, it has contributed to, if not triggered, a similar epidemic in wildlife known as chronic wasting disease (CWD). CWD is further confirmation of the transmissibility of prion disease. So is CWD and mad cow disease.
Unfortunately, connecting these dots is not difficult. Failure to connect these dots on behalf of public health is criminal. Prion disease is impossible to stop, which makes pathway management (public exposure) vital. Caregivers, family members and billions of innocent people are caught in the crossfire of this explosive epidemic.
The Prion Pandemic
Prions (PREE-ons) are a deadly and unstoppable form of protein that migrates, mutates, multiplies and kills with unparalleled efficiency. Prions cause fatal neurodegenerative disease in humans and other mammals by converting the cellular version of prion protein into a toxic form that erodes the brain and body. Prion disease often is described as a wasting disease that causes a loss of body mass and brain mass.
We know prion disease by other names, which helps cloak the gravity of the overall problem. As this article will explain, humans experience prion disease primarily as Alzheimer’s, Parkinson’s, Creutzfeldt-Jakob and Huntington diseases. In livestock, prion disease has been coined as mad cow disease. In deer, elk, moose and reindeer, prion disease is often referred to as chronic wasting disease. Prion disease has been found in camels, dolphins, elephants, mink, cats and many other species. The suggestion of a reliable species barrier against thousands, if not millions, of mutations is ludicrous.
Regardless of the species impacted, prion disease causes memory loss, impaired coordination, abnormal movements and overall wasting of the mind and body. Victims become prion incubators and distributors. So do wastewater treatment plants.
Governments and industry are working diligently to keep prion disease off the public radar. The epidemic will persist. A cure does not exist. Ignoring the truth is making it worse.
Despite millions of deaths annually, experts suggest that the prevalence of Alzheimer’s disease (and Parkinson’s disease) will quadruple by 2050, if not sooner. In the United States, for example, deaths from Alzheimer’s disease increased 71 percent from 2000 to 2013. During that time, deaths from heart disease decreased 14 percent and deaths from cancer also have been on the decline.
A groundbreaking study suggests that Alzheimer’s disease actually causes six times more deaths than official statistics indicate. In reality, the study said that Alzheimer’s disease was the underlying cause in more than 500,000 deaths (2010). These deaths were often listed as pneumonia or other complications caused by Alzheimer’s disease. Those numbers make Alzheimer’s disease the third-leading cause of death in the United States, behind heart disease and cancer. Researchers at the Rush University Medical Center in Chicago published their results in 2013 in the journal Neurology. If we could make an accurate guess of how many additional deaths were people who had Alzheimer’s disease, but were never diagnosed, statistics would surge even higher.
Until recently, few have considered the possibility that Alzheimer’s disease is a transmissible disease. This denial and misinformation has been fueling a public health crisis around the world for years.
Dr. Stanley Prusiner, an American neuroscientist from the University of California at San Francisco, earned a Nobel Prize in 1997 for discovering and characterizing prions and prion disease. President Obama awarded Prusiner the National Medal of Science in 2010 to recognize the importance of his research. Important reforms to policies to protect public health, however, have been elusive.
“Alzheimer’s disease (AD) is the most common neurodegenerative disease in humans and will pose a considerable challenge to healthcare systems in the coming years,” Prusiner said.
Prusiner’s most recent study confirms that Alzheimer’s disease is a prion disease, which means that millions of people with Alzheimer’s disease (and millions who died ahead of them) are highly infectious. Prion diseases are fatal, transmissible spongiform encephalopathies (TSEs) that affect humans and other animals.
“I believe this shows beyond a shadow of a doubt that amyloid beta and tau are both prions, and that Alzheimer’s disease is a double-prion disorder in which these two rogue proteins together destroy the brain,” said Stanley Prusiner, MD, the study’s senior author and director of the UCSF Institute for Neurodegenerative Diseases, part of the UCSF Weill Institute for Neurosciences. “The fact that prion levels also appear linked to patient longevity should change how we think about the way forward for developing treatments for the disease. We need a sea change in Alzheimer’s disease research, and that is what this paper does. This paper might catalyze a major change in AD research.”
Alzheimer’s disease is currently defined based on the presence of toxic protein aggregations in the brain known as amyloid plaques and tau tangles, accompanied by cognitive decline and dementia. New evidence that active Aß and tau prions could be driving the disease – published May 1, 2019 in Science Translational Medicine — could lead researchers to explore new therapies that focus on prions directly. Hopefully, it will lead to many reforms to safeguard public health.
“I learned that scrapie (sheep), Creutzfeldt-Jakob disease and kuru had all been shown to be transmissible by injecting extracts of diseased brains into the brains of healthy animals,” Prusiner said. Prusiner basically repeated the process with brain samples from those who died of Alzheimer’s disease to complete his latest study.
Prions + Pathways = Victims
Prion disease is highly contagious, incurable and fatal. Despite all of the smoke and mirrors, prion disease is prion disease. It’s killing more and more mammals, including humans, every year. The hype about species barriers is ridiculous, reckless and irresponsible.
Unfortunately, the CDC quietly took prions off the highly regulated list of select agents because the regulation criminalized entire industries and several reckless practices. It also opened the floodgates to prion mismanagement.
Victims should be quarantined because prions are in the milk, urine, feces, blood, saliva, mucus, skin and cell tissue of all victims–all human byproducts that are washed, dumped, or flushed down sinks and toilets. One can assume that the waste is extra infectious when it comes from funeral homes, nursing homes, hospitals, dental offices, veterinarians, slaughterhouses and some laboratories.
“There has been a resurgence of this sort of thinking, because there is now real evidence of the potential transmissibility of Alzheimer’s disease,” says Thomas Wiesniewski M.D. a prion and Alzheimer’s disease researcher at New York University School of Medicine.
A study published in the journal Nature adds to the evidence about the transmissibility of Alzheimer’s disease between people. A second study by the same scientist in early 2016 supports the claim. All logic says that humans also are passing prion disease to livestock and wildlife. Likewise, animals are transmitting prion disease back to humans via meat, milk and other pathways.
Unfortunately, the implications are profound. Prion pathways threaten each and every person on the planet. The greatest prion pathway in the world is human sewage. It’s contaminating food and water supplies with deadly prions now. It’s been going on for years, but it’s worse than ever now.
Claudio Soto, PhD, professor of neurology at the University of Texas Medical School in Houston, and his colleagues confirmed the presence of prions in urine. Prions infect the entire body and all bodily fluids of its victims, including blood, mucus and saliva. Caregivers for anyone with neurodegenerative disease beware.
“Our findings open the possibility that some of the sporadic Alzheimer’s disease cases may arise from an infectious process, which occurs with other neurological diseases such as mad cow disease and its human form, Creutzfeldt-Jakob disease,” said Soto. “The underlying mechanism of Alzheimer’s disease is very similar to the prion diseases. It involves a normal protein that becomes misshapen and is able to spread by transforming good proteins to bad ones. The bad proteins accumulate in the brain, forming plaque deposits (and inflammation), which kill neurons.”
Misfolded alpha-synuclein and tau protein associated with Parkinson’s disease and Alzheimer’s disease, respectively, have been reported in skin tissues of patients with these conditions. Skin could serve as a screen for early diagnosis, but also for monitoring the accumulation of the misfolded proteins in the brain of these neurodegenerative diseases. Unfortunately, skin (and other bodily tissues) is a potential pathway of transmission.
Prions are such a formidable threat that the U.S. government enacted the Bioterrorism Preparedness and Response Act of 2002, which included a provision to halt research on prions in all but two specialized laboratories.
The U.S. government initially classified prions as select agents that pose an extreme risk to food, water and health systems. Today, governments don’t regulate prions at all.
As such, millions of caregivers are being misinformed, misguided and exposed to an aggressive prion disease. So are friends and family. Unfortunately, Prusiner’s science is being ignored and we all are facing a public health disaster because of the negligence and reckless disregard for public health regarding prions.
“My latest study shows beyond a shadow of a doubt that amyloid beta and tau are both prions, and that Alzheimer’s disease is a double-prion disorder in which these two rogue proteins together destroy the brain,” said Prusiner. “The fact that prion levels also appear linked to patient longevity should change how we think about developing treatments for the disease. We need a sea change in Alzheimer’s disease research, and that is what this paper does. This paper might catalyze a major change in AD research.”
Prusiner claims that all forms of TSE are caused by infectious prions. Prusiner’s most recent study confirms that Alzheimer’s disease is a prion disease, which means that millions of people with Alzheimer’s disease (and millions who died ahead of them) are highly infectious. Alzheimer’s disease is currently defined based on the presence of toxic protein aggregations in the brain known as amyloid plaques and tau tangles, accompanied by cognitive decline and dementia.
“I learned that scrapie, Creutzfeldt-Jakob disease and kuru had all been shown to be transmissible by injecting extracts of diseased brains into the brains of healthy animals,” Prusiner said.
Prusiner basically repeated the process with brain samples from those who died of Alzheimer’s disease to complete his latest study. Sure enough, healthy animals contracted Alzheimer’s disease.
A previous study published in the journal Nature adds to the evidence about the transmissibility of Alzheimer’s disease between people. A second study by the same scientist in 2016 supports the claim. Unfortunately, the implications are profound. Prion pathways threaten each and every person on the planet.
Prion Disease A Spectrum Disease
TSE is a spectrum disease that varies in severity and symptoms. The diagnosis depends on which region of the brain is impacted first and by what prion mutation. Few cases of prion disease are identical in terms of symptoms and pathology.
According to neuroscientists Dr. Laura Manuelidis, at least 25 percent of Alzheimer’s diagnoses are actually Creutzfeldt-Jakob disease (CJD), which is further up the prion spectrum. CJD, without dispute, is extremely infectious to caregivers and loved ones.
When the presenting symptom is memory loss in a human, the diagnoses flow along the following chart.
In humans, the prion spectrum primarily includes Alzheimer’s disease, Parkinson’s disease and CJD–the most aggressive version. Since there are thousands, if not millions of mutations of prions that can impact various regions of the brain, few cases are the same.
The difference between these diseases is very slight and often indistinguishable to neurologists. Due to the similarities, millions of people have CJD, but are diagnosed by their doctors with Alzheimer’s disease. CJD is clearly an aggressive prion disease, but mismanaged on most levels. If we hold to conventional wisdom, where along the spectrum does prion disease become transmissible?
There is no reason to think that anyone on the prion spectrum isn’t a walking time bomb as they expose everyone and everything in their path. It’s impossible to sterilize anything that they touch.
Adding to the madness, CJD has not been declared a reportable disease across most nations, so tracking it and containing it from an epidemiological standpoint is impossible. Likewise, Alzheimer’s disease rarely makes it to the death certificate as the primary cause of death. This is the silent pandemic.
Meanwhile, abnormal proteins also are associated with autism, which began spiking in the 1980s. It’s worse than ever today, but there is no national screening plan in most countries. In fact, it appears that age is the biggest difference between the neurodegenerative disease spectrum and autism spectrum disorders (neurodevelopmental). Both share common environmental causes and pathologies. Plus, CJD is taking the lives of more and more young adults and adolescents. The lines are clearly blurring with time and truth.
Claudio Soto and his colleagues also confirmed the presence of prions in the urine of those who have prion disease. Prions actually infect the entire body and all bodily fluids of its victims. Caregivers beware.
The greatest prion pathway in the world is human sewage. Wastewater treatment plants are collecting points for prions from infected humans. The sewage treatment process can’t stop prions from migrating, mutating and multiplying before being discharged into the environment where they strengthen with each mutation.
Wastewater treatment plants collect millions of gallons of this infectious waste every day. They are incapable of stopping prions, which makes these facilities incubators for prions. It’s a centralized place where they can incubate and mutate before being recycled into reusable water or fertilizer (biosolids). Industrialized nations and developing nations alike are poisoning us all with sewage laced with prions, carcinogens and much more. Because of the prion factor alone, all by-products and discharges from wastewater treatment plants are infectious waste, which are contributing to the global epidemic of neurodegenerative disease among humans, wildlife, sea mammals and livestock.
The U.S. Environmental Protection Agency (EPA) has confirmed that prions are in sewage and that there has been no way to detect them or stop them. As such, the EPA has never issued guidance on prion management within wastewater treatment plants. Unfortunately, the EPA’s risk assessment on sewage sludge (biosolids) was prepared before the world of science knew about prions. The agency clung to its antiquated sludge rule crafted back in the dark ages until recently. All along, it considered prions as an “emerging contaminant of concern.”
The risk assessments prepared by the U.S. EPA for wastewater treatment and sewage sludge are flawed and current practices of recycling this infectious waste are fueling a public health disaster. Many risks are not addressed, including prions and radioactive waste. They don’t mention prions or radiation because there is no answer. Most nations are making the same mistake. We’re dumping killer proteins on crops, parks, golf courses, gardens, ski areas, school grounds and beyond. Wind, rain and irrigation spread these contaminants and many more throughout our communities and watersheds.
Meanwhile, its outdated risk assessments are still in use. They are promoting a public health disaster. The neurotoxins found in sewage, including heavy metals, also are contributing to the global spike in autism, which follows the same timing and trajectory as the spike in neurodegenerative diseases.
“Since it’s unlikely that the sewage treatment process can effectively stop prions, adopting measures to prevent the entry of prions into the sewer system is advisable,” said the Toronto Department of Health, November 2004.
Once unleashed on the environment, prions remain infectious. They migrate, mutate and multiply as they infect crops, water supplies, wildlife, livestock, sea mammals and humans. According to prion researcher Joel Pedersen at the University of Wisconsin, prions in soil become up to 680 times more infectious. From there, they migrate, mutate and multiply. It’s a real world version of Pandora’s Lunchbox.
“Our results suggest that if prions enter municipal wastewater treatment systems, most prions would bond to sewage sludge, survive anaerobic digestion, and be present in treated biosolids,” Pedersen said. “Land application of biosolids containing prions represents a route for their introduction into the environment. I emphasize the importance of keeping prions out of municipal wastewater treatment systems.”
Pedersen also found that sewage treatment does not inactivate prions. Therefore, prions are lethal, mutating, migrating and multiplying everywhere sewage (biosolids) is dumped.
Prions could end up in sewage treatment plants via slaughterhouses, hospitals, dental offices and mortuaries just to name a few of the pathways. The disposal of sludge represents the greatest risk of spreading prion contamination in the environment. Plus, we know that pathogens, pharmaceutical residue and chemical pollutants found in sewage sludge are taken up by plants and vegetables.”
Thanks to the mismanagement of infectious waste, including sewage, the animal world is contracting prion disease from humans. They also are passing it among themselves via their own bodily fluids. When it comes to prion disease, species barriers are a myth.
Unfortunately, prions linger in the environment, homes, hospitals, nursing homes, dental offices and beyond infinitely. Prions defy all attempts at sterilization and inactivation. If they can’t stop prions in the friendly and sterile confines of an operating room, they can’t stop them in the wastewater treatment plant.
Dr. Soto also confirmed that plants uptake prions from the soil and water. The plants become fatally infectious to those who consume them. Even wildlife and sea mammals are contracting brain disease from people because of the dumping of infectious waste on farms, ranches and forests. Humans, wildlife and livestock are vulnerable to prion disease via crops and plants grown on land treated with infectious waste (sewage sludge, biosolids and reclaimed wastewater).
Crops for humans and livestock grown in sewage sludge absorb prions and other neurotoxins. They become infectious. We’re all vulnerable to neurotoxins right now due to widespread denial and mismanagement. It’s time to stop the land application of sewage sludge (LASS) in all nations. Safer alternatives exist.
Prion disease is highly contagious, incurable and fatal. Despite all of the smoke and mirrors, prion disease is prion disease. It’s killing more and more mammals, including humans, every year. The hype about species barriers is ridiculous, reckless and irresponsible.
Victims should be quarantined because prions are in the urine, feces, blood, saliva, mucus, skin and cell tissue of all victims–all human byproducts that are washed, dumped, or flushed down sinks and toilets. One can assume that the waste is extra infectious with prions when it comes from funeral homes, nursing homes, hospitals, dental offices, veterinarians, slaughterhouses and some laboratories.
Wastewater treatment plants (WWTPs) in the United States generate approximately eight million dry tons of biosolids each year. Biosolids is a green washed name for sewage sludge—the most toxic and variable part of the wastewater treatment process. Sewage sludge was deemed too toxic to dump at sea, but the EPA gave its blessing to dump millions of tons of this toxic waste on farms, ranches, parks, school grounds, golf courses and gardens.
Since biosolids are rich in plant nutrients, farmers, landscapers, and homeowners use about 50 percent of the annual production of biosolids as fertilizer. Because a variety of pharmaceuticals and other household chemicals have been found in the wastewater discharged from WWTPs, questions have been raised about the presence of these chemicals in biosolids. To help answer the questions the scientists purchased or obtained nine different commercially or publicly available biosolids and analyzed them for 87 organic chemicals found in cleaners, personal care products, pharmaceuticals, and other products. They found:
- Fifty-five of the 87 organic chemicals measured were detected in at least one of the nine biosolids collected, with as many as 45 chemicals found in a single sample.
- Twenty-five of the chemicals were present in every biosolids sample including compounds that are pharmaceutically and hormonally active, such as an antimicrobial disinfectant (triclosan), a musk fragrance (tonalide), an antihistamine (diphenhydramine), and an antiepileptic drug (carbamazepine).
- The biosolids were more similar than they were different, even though they were produced by a variety of treatment processes from plants serving vastly different sized cities and towns. The types of contaminants and their relation to each other did not vary greatly between the biosolids tested.
In addition to prions, this comprehensive examination indicates that biosolids have high concentrations of these emerging contaminants, so the risks associated with biosolids as a fertilizer are numerous.
Unfortunately, prions linger in the environment, homes, hospitals, nursing homes, and dental offices and beyond infinitely. Prions defy all attempts at sterilization and inactivation. If they can’t stop prions in the friendly and sterile confines of an operating room, they can’t stop them in the wastewater treatment plant. That’s one of the reasons why several countries, have banned the land application of biosolids because of concerns for human, livestock and wildlife.
“Our findings open the possibility that some of the sporadic Alzheimer’s disease cases may arise from an infectious process, which occurs with other neurological diseases such as mad cow disease and its human form, Creutzfeldt-Jakob disease,” said Soto. “The underlying mechanism of Alzheimer’s disease is very similar to the prion diseases. It involves a normal protein that becomes misshapen and is able to spread by transforming good proteins to bad ones. The bad proteins accumulate in the brain, forming plaque deposits (and inflammation), which kill neuron cells in Alzheimer’s disease.”
Since the critical research on Alzheimer’s disease and infectivity have been blocked, let’s build on the research completed by Prusiner, Soto and Pederson. If prions are as infectious as stated, and if the Alzheimer’s pandemic is widespread as stated, WWTPs around the world are now collectors, incubators and distributors of prions. Since there has not been any research based on biosolids and prion infectivity to livestock and wildlife, we should be able to find examples of prion disease in animals in regions where biosolids have been spread on crops, pastures, forests, parks, golf courses and beyond.
In livestock, prion disease is known as mad cow disease. Since the United States and others have blocked all attempts to implement a system to test cattle for prion disease, we can’t use livestock as a bio-indicator, but we can use wildlife.
In wildlife, prion disease has been most commonly coined as chronic wasting disease (CWD). Unfortunately, CWD is a living laboratory experiment that is out of control across the majority of the United States. CWD is now across North America and Norway. It’s also in camels in the Persian Gulf and Northern Africa.
CWD is now emerging in other nations that dump sewage on land. It will get worse until the wildlife populations are virtually depleted. Of course, the sick animals are just proverbial canaries in a coalmine. Their decline mirrors our own.
In my opinion, there isn’t a single explanation for prion disease. We all have healthy prions in our bodies. When prions start misfolding, all hell breaks loose. It’s the neurological equivalent to cancer, radiation poisoning and more all rolled into one threat that consumes the mind and body. Unfortunately, each victim becomes a walking time bomb as they expose everyone and everything around them. Prion aversion is our only hope. Answers begin with the truth.
Preview and order the eBook now to defend yourself and your family. There is no cure for prion disease, but smart nutrition can ease the symptoms. Smart nutrition also can help you and your family avert neurodegenerative disease.
Gary Chandler is a prion expert. He is the CEO of Crossbow Communications, author of several books and producer of documentaries about health and environmental issues around the world. Chandler is connecting the dots to the global surge in neurodegenerative disease, including Alzheimer’s disease, Parkinson’s disease, Creutzfeldt-Jakob disease, chronic wasting disease and other forms of prion disease. The scientific name for prion disease is transmissible spongiform encephalopathy.